Common genetic variants of homocysteine metabolism in ischemic stroke: a case-control study

Eur J Neurol. 2005 Aug;12(8):614-8. doi: 10.1111/j.1468-1331.2005.01045.x.

Abstract

Hyperhomocysteinemia is a risk factor for ischemic stroke. We investigated five functional polymorphisms involved in homocysteine metabolism in each 159 stroke patients and controls. The folate-sensitive polymorphism methylenetetrahydrofolate reductase (MTHFR) c. 677 C > T (A222V) referred a non-significant risk of ischemic stroke (odds ratio: 1.20) in all patients, and homozygosity for MTHFR c. 677 C > T was associated with an earlier onset of stroke selectively in patients younger than 60 years (38 +/- 3 years vs. 45 +/- 1 years; P = 0.043). This study suggests that the investigated polymorphisms are no major risk factors for stroke, although MTHFR c. 677 C > T could be a minor factor of vulnerability especially in young patients (TT genotype), which might be helpful for the clinical work-up of stroke cases and for preventive dietary strategies.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Age Factors
  • Aged
  • Case-Control Studies
  • Confidence Intervals
  • DNA Mutational Analysis / methods
  • Female
  • Gene Frequency
  • Genetic Variation*
  • Genotype
  • Homocysteine / genetics*
  • Homocysteine / metabolism*
  • Humans
  • Male
  • Methylenetetrahydrofolate Reductase (NADPH2) / genetics*
  • Middle Aged
  • Odds Ratio
  • RNA, Messenger / biosynthesis
  • Retrospective Studies
  • Reverse Transcriptase Polymerase Chain Reaction / methods
  • Stroke / genetics
  • Stroke / metabolism*

Substances

  • RNA, Messenger
  • Homocysteine
  • Methylenetetrahydrofolate Reductase (NADPH2)