Lack of MHC-II expression in activated mouse T cells correlates with DNA methylation at the CIITA-PIII region

Immunogenetics. 2005 Nov;57(10):795-9. doi: 10.1007/s00251-005-0051-8. Epub 2005 Nov 8.

Abstract

In contrast to activated human T cells, activated mouse T cells fail to express MHC class II molecules (MHC-II) at their cell surface. This is because mouse T cells hardly produce mRNA encoding the MHC-II molecules I-A and I-E, due to severely impaired expression levels upon T-cell activation of the mhc2ta gene, encoding the class II transactivator (CIITA). In humans, activated T cells express exclusively the CIITA promoter III (CIITA-PIII) isoform, which results in cell surface expression of all MHC-II isotypes (HLA-DR, -DP and -DQ). In this study, we demonstrate that methylation of CIITA-PIII contributes to the failure of mouse T cells to transcribe the mhc2ta and the resulting I-A/E genes, explaining the lack of I-A/E molecule expression at the cell surface following activation.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line
  • DNA Methylation*
  • Gene Expression Regulation
  • Histocompatibility Antigens Class II / biosynthesis*
  • Histocompatibility Antigens Class II / genetics
  • Humans
  • Lymphocyte Activation
  • Mice
  • Nuclear Proteins / genetics*
  • Nuclear Proteins / immunology
  • T-Lymphocytes / immunology*
  • T-Lymphocytes / metabolism
  • Trans-Activators / genetics*
  • Trans-Activators / immunology

Substances

  • Histocompatibility Antigens Class II
  • MHC class II transactivator protein
  • Nuclear Proteins
  • Trans-Activators