The regulated expression of c-IAP1 and c-IAP2 during the rat seminiferous epithelial cycle plays a role in the protection of germ cells from Fas-mediated apoptosis

Mol Cell Endocrinol. 2005 Dec 21;245(1-2):111-20. doi: 10.1016/j.mce.2005.11.004. Epub 2005 Dec 15.

Abstract

The inhibitor of apoptosis proteins, c-IAP1 and c-IAP2, are highly expressed in rat testis and potentially play a regulatory role in testicular apoptosis. To better understand their functions during spermatogenesis, we have analyzed their spatio-temporal distribution in rat testis, how their expression is controlled by the paracrine stem-cell factor (SCF) and how they affect Fas-mediated apoptosis. Both c-IAP1 and c-IAP2 showed cycles of transcriptional expression, throughout the seminiferous epithelial cycle. c-IAP1 protein showed a diffuse nuclear distribution in type B spermatogonia, preleptotene, leptotene, and zygotene spermatocytes. In pachytene spermatocytes, c-IAP1 colocalized with SUMO-1 in the XY-body. c-IAP2 protein was cytoplasmic in spermatocytes, from stage VI pachytene onwards, round spermatids, elongated spermatids and Leydig cells. Its expression was upregulated by SCF. Inhibition of IAP activity resulted in a greater sensitivity of germ cells to Fas-mediated apoptosis. These results suggest an important role for IAPs in the regulation of spermatogenic apoptosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis* / drug effects
  • Caspases / physiology
  • Cell Cycle / genetics
  • Cell Cycle / physiology
  • Enzyme Activation
  • Gene Expression Regulation*
  • Immunohistochemistry
  • Inhibitor of Apoptosis Proteins / antagonists & inhibitors
  • Inhibitor of Apoptosis Proteins / genetics*
  • Inhibitor of Apoptosis Proteins / metabolism
  • Male
  • Rats
  • Rats, Sprague-Dawley
  • Seminiferous Epithelium / cytology
  • Seminiferous Epithelium / physiology*
  • Spermatogenesis / genetics
  • Spermatogenesis / physiology
  • Spermatozoa / cytology
  • Spermatozoa / physiology*
  • Stem Cell Factor / physiology
  • Testis / metabolism
  • Up-Regulation / drug effects
  • fas Receptor / pharmacology
  • fas Receptor / physiology*

Substances

  • Inhibitor of Apoptosis Proteins
  • Stem Cell Factor
  • fas Receptor
  • Caspases