A novel presenilin 1 L166H mutation in a pseudo-sporadic case of early-onset Alzheimer's disease

Neurol Sci. 2005 Dec;26(5):349-50. doi: 10.1007/s10072-005-0499-1.

Abstract

We report a 44-year-old woman presenting at 33 years with memory loss, followed by progressive dementia. Her family history was negative for dominant genetic disorders at high penetrance. Analysis of presenilin-1 gene revealed a missense mutation at codon 166, leading to the substitution from leucine to histidine. The mutation occurs in the third transmembrane domain of presenilin-1, at the position of two different mutations previously described, associated with an atypical phenotype. The present case has two implications: (1) mutations of presenilin-1 have to be searched also in apparently sporadic cases of dementia beginning in the third decade of life; (2) as yet unidentified factors, besides the gamma-secretase complex, influence the phenotype of presenilin-1 mutations.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Alzheimer Disease / genetics*
  • DNA Mutational Analysis / methods
  • Female
  • Histidine / genetics*
  • Humans
  • Leucine / genetics*
  • Membrane Proteins / genetics*
  • Mutation*
  • Presenilin-1

Substances

  • Membrane Proteins
  • PSEN1 protein, human
  • Presenilin-1
  • Histidine
  • Leucine