Abstract
Seven families with six different SPG3A mutations were identified among 106 with autosomal dominant hereditary spastic paraplegia (HSP). Two mutations were novel (T162P, C375R). SPG3A was twice as frequent as SPG4 in patients with onset before age 10 years (31.8%). Later onset was not observed. The phenotype was pure HSP, but disease duration was longer than in non-SPG3A/SPG4 patients, leading ultimately to greater handicap.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adolescent
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Adult
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Age of Onset
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Aged
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Brain / pathology
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Brain / physiopathology
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DNA Mutational Analysis
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Family Health
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GTP Phosphohydrolases / genetics*
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GTP-Binding Proteins
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Genetic Predisposition to Disease / genetics*
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Genetic Testing
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Humans
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Male
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Membrane Proteins
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Middle Aged
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Mutation / genetics*
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Peripheral Nerves / pathology
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Peripheral Nerves / physiopathology
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Phenotype
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Spastic Paraplegia, Hereditary / diagnosis
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Spastic Paraplegia, Hereditary / genetics*
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Spastic Paraplegia, Hereditary / physiopathology
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Wallerian Degeneration / diagnosis
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Wallerian Degeneration / genetics
Substances
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Membrane Proteins
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ATL1 protein, human
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GTP Phosphohydrolases
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GTP-Binding Proteins