Abstract
Apolipoprotein B mRNA-editing enzyme catalytic subunit 2 (APOBEC2) is a member of the nucleic-acid-editing enzymes. However, the physiological function of APOBEC2 remains unclear. We demonstrate that APOBEC2 expression is strongly enhanced in response to both tumour necrosis factor-alpha (TNF-alpha) and interleukin-1beta. Inhibition of NF-kappaB activation invariably blocks TNF-alpha-induced APOBEC2 expression. The promoter region of APOBEC2 contains functional NF-kappaB response elements in the 5' untranslated region of the gene at -625/-616. These results show that APOBEC2 expression is regulated by pro-inflammatory cytokines via NF-kappaB activation and suggest a possible role of APOBEC2 in the pathophysiology of hepatic inflammation.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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APOBEC Deaminases
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Cell Line, Tumor
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Cytidine Deaminase / biosynthesis*
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Cytidine Deaminase / genetics
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Gene Expression Regulation* / drug effects
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Gene Expression Regulation* / genetics
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Hepatitis / enzymology
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Hepatitis / genetics
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Hepatitis / pathology
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Hepatocytes / enzymology*
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Humans
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Interleukin-1 / metabolism
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Interleukin-1 / pharmacology
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Muscle Proteins / biosynthesis*
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Muscle Proteins / genetics
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NF-kappa B / metabolism*
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RNA Editing* / drug effects
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RNA Editing* / genetics
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Response Elements / genetics
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Tumor Necrosis Factor-alpha / metabolism
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Tumor Necrosis Factor-alpha / pharmacology
Substances
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Interleukin-1
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Muscle Proteins
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NF-kappa B
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Tumor Necrosis Factor-alpha
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APOBEC Deaminases
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APOBEC2 protein, human
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Cytidine Deaminase