Immunological evaluation of late complement component-deficient individuals

Clin Immunol Immunopathol. 1992 Aug;64(2):98-105. doi: 10.1016/0090-1229(92)90186-r.

Abstract

The possible contribution of additional immunologic variables to the susceptibility of late complement component-deficient individuals to meningococcal disease has not been systematically examined in previous studies. Thus, we studied three groups of patients: (1) 24 healthy individuals, (2) 8 complement-sufficient individuals with a history of recurrent bacterial meningitis, and (3) 19 complement-deficient individuals with prior meningococcal infection. No statistical differences were noted among the three groups for the following parameters: the absolute number and the percentage of lymphocytes; CD3+, CD4+, CD8+, CD20+, and CD16+ cells; and the CD4+/CD8+ ratio. The concentration of C4 and circulating immune complexes was also similar among the groups. The concentrations of IgG, IgM, and IgA were slightly, but significantly, decreased in the complement-deficient individuals. Of interest, the coefficient of spontaneous and lipopolysaccharide-stimulated activation of neutrophils was significantly depressed in the deficient individuals. We hypothesize that the terminal complement components may participate in maximal neutrophil activation.

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Antigen-Antibody Complex / metabolism
  • Child
  • Complement C4 / metabolism
  • Complement C5 / deficiency
  • Complement C6 / deficiency
  • Complement C7 / deficiency
  • Complement C8 / deficiency
  • Complement C9 / deficiency
  • Complement System Proteins / deficiency*
  • Complement System Proteins / immunology
  • Female
  • Humans
  • Immunoglobulins / blood
  • Leukocyte Count
  • Male
  • Middle Aged
  • Neutrophils / metabolism

Substances

  • Antigen-Antibody Complex
  • Complement C4
  • Complement C5
  • Complement C6
  • Complement C7
  • Complement C8
  • Complement C9
  • Immunoglobulins
  • Complement System Proteins