Loss of PTEN is associated with progression to androgen independence

Prostate. 2006 Jun 15;66(9):895-902. doi: 10.1002/pros.20411.

Abstract

Background: Progression to a lethal androgen-independent (AI) stage of advanced prostate cancer is a critical clinical obstacle limiting patient survival. PTEN inactivation is frequently observed in advanced prostate cancer and correlates with a poor prognosis. However, the functional significance of PTEN inactivation in AI progression has not been demonstrated.

Methods: PTEN expression was examined in benign, hormone naïve and AI human prostate cancer specimens, and in recurrent AI Shionogi tumors. The effect of antisense oligonucleotide (ASO)-mediated PTEN downregulation in AI progression of the Shionogi tumor model was determined.

Results: Significantly reduced PTEN expression was observed in AI versus benign and hormone naïve prostate tumors. Seven of 14 AI Shionogi tumors exhibited marked downregulation or complete loss of PTEN. ASO-mediated PTEN inhibition reduced androgen-withdrawal induced regression of Shionogi tumors and accelerated AI progression.

Conclusions: These data suggest that PTEN inactivation may play a role in progression to androgen independence.

Publication types

  • Comparative Study

MeSH terms

  • Androgens / analysis
  • Androgens / physiology*
  • Animals
  • Blotting, Northern
  • Blotting, Western
  • Disease Progression
  • Down-Regulation
  • Gene Expression Regulation, Neoplastic / drug effects
  • Genes, Suppressor / physiology
  • Humans
  • Male
  • Mice
  • Mice, Inbred Strains
  • Neoplasm Staging
  • Oligonucleotides, Antisense / pharmacology
  • PTEN Phosphohydrolase / genetics*
  • PTEN Phosphohydrolase / physiology*
  • Phosphatidylinositol 3-Kinases / physiology
  • Prognosis
  • Prostatic Neoplasms / chemistry
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / physiopathology*
  • Signal Transduction / physiology

Substances

  • Androgens
  • Oligonucleotides, Antisense
  • Phosphatidylinositol 3-Kinases
  • PTEN Phosphohydrolase
  • PTEN protein, human