Dibutyryl cyclic AMP induces formyl peptide receptor expression and chemotactic responses in a human eosinophilic cell line, EoL-1

Exp Hematol. 1991 Sep;19(8):823-8.

Abstract

We investigated actin polymerization and the increase of cytosolic free calcium concentration ([Ca2+]i) in a human eosinophilic leukemia cell line, EoL-1, in response to stimulation with chemotactic factors; we also investigated the effect of dibutyryl cyclic AMP (dbcAMP) on the responses. EoL-1 cells under normal culture conditions did not show either actin polymerization or an increase in [Ca2+]i when stimulated with formyl-methionyl-leucyl-phenylalanine (fMLP). Expression of formyl peptide receptors was not detectable on untreated EoL-1 cells, either. Dibutyryl cAMP induced the expression of formyl peptide receptors and the responsiveness to fMLP. The responsiveness of EoL-1 cells to the complement fragment C5a and platelet-activating factor (PAF) was also induced or enhanced by dbcAMP. The growth of EoL-1 cells was decreased and the proportion of cells in the G0/G1 phase of the cell cycle was increased by the treatment of EoL-1 cells with dbcAMP. The proportion of eosinophilic granule-containing cells and the content of eosinophil cationic protein (ECP) in EoL-1 cells was also increased when they were stimulated with dbcAMP for a longer period. The responsiveness of EoL-1 cells to fMLP, C5a, and PAF was shown to be regulated independently. EoL-1 cells and dbcAMP seem to be useful for examining chemotactic receptor expression and its signal transduction mechanisms.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / metabolism
  • Bucladesine / pharmacology*
  • Butyrates / pharmacology
  • Butyric Acid
  • Calcium / physiology
  • Cell Differentiation / drug effects
  • Cell Division / drug effects
  • Cell Line
  • Chemotaxis, Leukocyte / drug effects*
  • Complement C5a / pharmacology
  • Eosinophils / cytology
  • Eosinophils / drug effects
  • Eosinophils / physiology*
  • Humans
  • N-Formylmethionine Leucyl-Phenylalanine / metabolism
  • Platelet Activating Factor / pharmacology
  • Polymers
  • Receptors, Formyl Peptide
  • Receptors, Immunologic / metabolism*
  • Time Factors

Substances

  • Actins
  • Butyrates
  • Platelet Activating Factor
  • Polymers
  • Receptors, Formyl Peptide
  • Receptors, Immunologic
  • Butyric Acid
  • N-Formylmethionine Leucyl-Phenylalanine
  • Bucladesine
  • Complement C5a
  • Calcium