Rituximab for steroid-refractory chronic graft-versus-host disease

Blood. 2006 Jul 15;108(2):756-62. doi: 10.1182/blood-2006-01-0233. Epub 2006 Mar 21.

Abstract

B cells may be implicated in the pathophysiology of chronic graft-versus-host disease (GVHD), as evidenced by antibody production against sex-mismatched, Y chromosome-encoded minor HLA antigens in association with chronic GVHD. We therefore designed a phase 1/2 study of anti-B-cell therapy with rituximab in steroid-refractory chronic GVHD. Twenty-one patients were treated with 38 cycles of rituximab. Rituximab was tolerated well, and toxicity was limited to infectious events. The clinical response rate was 70%, including 2 patients with complete responses. Responses were limited to patients with cutaneous and musculoskeletal manifestations of chronic GVHD and were durable through 1 year after therapy. The median dose of prednisone among treated subjects fell from 40 mg/day to 10 mg/day, 1 year after rituximab therapy (P < .001). A chronic GVHD symptom score improved in the majority of treated patients. Antibody titers against Y chromosome-encoded minor HLA antigens fell and remained low, whereas titers against infectious antigens (EBV, tetanus) remained stable or rose during the treatment period. We conclude that specific anti-B-cell therapy with rituximab may be beneficial for patients with steroidrefractory chronic GVHD. This trial was registered at www.clinicaltrials.gov as #NCT00136396.

Publication types

  • Clinical Trial, Phase I
  • Clinical Trial, Phase II
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Antibodies, Monoclonal / administration & dosage*
  • Antibodies, Monoclonal, Murine-Derived
  • B-Lymphocytes / drug effects
  • B-Lymphocytes / pathology
  • Chronic Disease
  • Drug Resistance*
  • Female
  • Graft vs Host Disease / drug therapy*
  • Graft vs Host Disease / pathology
  • Humans
  • Male
  • Middle Aged
  • Musculoskeletal Diseases / drug therapy
  • Musculoskeletal Diseases / etiology
  • Prednisone / pharmacology
  • Rituximab
  • Salvage Therapy / methods
  • Skin Diseases / drug therapy
  • Skin Diseases / etiology
  • Steroids / pharmacology*

Substances

  • Antibodies, Monoclonal
  • Antibodies, Monoclonal, Murine-Derived
  • Steroids
  • Rituximab
  • Prednisone

Associated data

  • ClinicalTrials.gov/NCT00136396