Detection of recurrent copy number loss at Yp11.2 involving TSPY gene cluster in prostate cancer using array-based comparative genomic hybridization

Cancer Res. 2006 Apr 15;66(8):4055-64. doi: 10.1158/0008-5472.CAN-05-3822.

Abstract

Prostate cancer is the second leading cause of cancer deaths among American men. The loss of Y chromosome has been frequently observed in primary prostate cancer as well as other types of cancer. Earlier, we showed that introduction of the human Y chromosome suppresses the in vivo tumorigenicity of the prostate cancer cell line PC-3. To further characterize the Y chromosome, we have developed a high-density bacterial artificial chromosome (BAC) microarray containing 178 BAC clones from the human Y chromosome. BAC microarray was used for array comparative genomic hybridization on prostate cancer samples and cell lines. The most prominent observation on prostate cancer specimens was a deletion at Yp11.2 containing the TSPY tandem gene array. Out of 36 primary prostate tumors analyzed, 16 (44.4%) samples exhibited loss of TSPY gene copies. Notably, we observed association between the number of TSPY copies in the blood and the incidence of prostate cancer. Moreover, PC-3 hybrids with an intact Yp11.2 did not grow tumors in nude mice, whereas PC-3 hybrids with a deletion at Yp11.2 grew tumors in nude mice.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Aged
  • Cell Cycle Proteins / genetics*
  • Cell Line, Tumor
  • Chromosomes, Human, Y / genetics*
  • Gene Dosage
  • Humans
  • Male
  • Middle Aged
  • Multigene Family
  • Neoplasm Staging
  • Nucleic Acid Hybridization
  • Oligonucleotide Array Sequence Analysis / methods
  • Oligonucleotide Array Sequence Analysis / standards
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / pathology
  • Reverse Transcriptase Polymerase Chain Reaction

Substances

  • Cell Cycle Proteins
  • TSPY1 protein, human