Mutation of waaC, encoding heptosyltransferase I in Campylobacter jejuni 81-176, affects the structure of both lipooligosaccharide and capsular carbohydrate

J Bacteriol. 2006 May;188(9):3273-9. doi: 10.1128/JB.188.9.3273-3279.2006.

Abstract

Campylobacter jejuni 81-176 lipooligosaccharide (LOS) is composed of two covalently linked domains: lipid A, a hydrophobic anchor, and a nonrepeating core oligosaccharide, consisting of an inner and outer core region. We report the isolation and characterization of the deepest rough C. jejuni 81-176 mutant by insertional mutagenesis into the waaC gene, encoding heptosyltransferase I that catalyzes the transfer of the first L-glycero-D-manno-heptose residue to 3-deoxy-D-manno-octulosonic residue (Kdo)-lipid A. Tricine gel electrophoresis, followed by silver staining, showed that site-specific mutation in the waaC gene resulted in the expression of a severely truncated LOS compared to wild-type strain 81-176. Gas-liquid chromatography-mass spectrometry and nuclear magnetic resonance spectroscopy showed that the waaC LOS species lacked all sugars distal to Kdo-lipid A. Parallel structural studies of the capsular polysaccharides of the wild-type strain 81-176 and waaC mutant revealed loss of the 3-O-methyl group in the waaC mutant. Complementation of the C. jejuni mutant by insertion of the wild-type C. jejuni waaC gene into a chromosomal locus resulted in LOS and capsular structures identical to those expressed in the parent strain. We also report here the presence of O-methyl phosphoramidate in wild-type strain 81-176 capsular polysaccharide.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bacterial Capsules / chemistry*
  • Bacterial Capsules / metabolism
  • Campylobacter jejuni / genetics
  • Campylobacter jejuni / metabolism*
  • Campylobacter jejuni / pathogenicity
  • Carbohydrate Metabolism
  • Carbohydrates / analysis
  • Carbohydrates / chemistry*
  • DNA, Bacterial
  • Electrophoresis, Gel, Two-Dimensional
  • Flavonoids
  • Gastroenteritis / etiology
  • Glycosyltransferases / genetics
  • Glycosyltransferases / metabolism*
  • Guillain-Barre Syndrome / etiology
  • Lipopolysaccharides / chemistry*
  • Lipopolysaccharides / metabolism
  • Magnetic Resonance Spectroscopy
  • Molecular Sequence Data
  • Mutagenesis, Insertional
  • Virulence

Substances

  • Carbohydrates
  • DNA, Bacterial
  • Flavonoids
  • Lipopolysaccharides
  • lipid-linked oligosaccharides
  • tricin
  • Glycosyltransferases
  • heptosyltransferase

Associated data

  • GENBANK/AY862985