Involvement of Sp-1 in the regulation of the Id-1 gene during trophoblast cell differentiation

Placenta. 2007 Feb-Mar;28(2-3):192-8. doi: 10.1016/j.placenta.2006.03.002. Epub 2006 Apr 25.

Abstract

Id-1, a member of the helix-loop-helix transcription factor family, inhibits the differentiation of Rcho-1 cells, which were derived from rat choriocarcinoma and consist of trophoblast stem cells. Id-1 is expressed at a high level in undifferentiated trophoblast stem cells and then down-regulated during early differentiation, and is thought to be a key regulator in the trophoblast giant-cell differentiation pathway. In this study, we analyzed the signaling mechanism regulating the high expression levels of Id-1 in undifferentiated Rcho-1 cells. Promoter deletion analysis revealed that a 31-bp sequence (Box-2 region), located between -200 and -169bp in the Id-1 promoter is necessary for the promoter activity. Electrophoretic mobility shift assays and DNA affinity precipitation assays showed that Box-2-binding activity was decreased during differentiation and that Sp-1 protein bound to this sequence. The protein level of Sp-1 was decreased during the differentiation. These results suggest that the Sp-1 protein level may regulate the Box-2-binding activity and the trophoblast giant-cell differentiation.

MeSH terms

  • Animals
  • Cell Differentiation / physiology*
  • Cell Line
  • Electrophoresis, Polyacrylamide Gel
  • Electrophoretic Mobility Shift Assay
  • Gene Expression Regulation
  • Inhibitor of Differentiation Protein 1 / genetics
  • Inhibitor of Differentiation Protein 1 / physiology*
  • Promoter Regions, Genetic
  • Rats
  • Signal Transduction / physiology
  • Sp1 Transcription Factor / physiology*
  • Trophoblasts / cytology
  • Trophoblasts / physiology*

Substances

  • ID1 protein, rat
  • Inhibitor of Differentiation Protein 1
  • Sp1 Transcription Factor