Liver dysfunction following splenectomy in idiopathic myelofibrosis: a study of 10 patients

Acta Haematol. 1991;85(4):184-8. doi: 10.1159/000204888.

Abstract

The hepatic abnormalities that developed after the splenectomy in 10 subjects with idiopathic myelofibrosis were analyzed. In all patients in whom a liver biopsy was performed during the splenectomy, extramedullary hematopoiesis was demonstrated, consisting of dysmorphic megakaryocytes primarily localized in the sinusoids, often accompanied by erythroid precursors. Following splenectomy, a significant increase in both the liver size and serum levels of alkaline phosphatase, bilirubin or gamma-glutamyl transpeptidase was found within 6 months, whereas no such increase was observed in the serum aspartate transaminase and alanine transaminase concentrations. In addition, 2 patients developed acute liver failure leading to death at 3 and 4 weeks from splenectomy, respectively. In contrast with these findings, no hepatic alterations were observed in 10 chronic myeloid leukemia patients who were also submitted to splenectomy.

MeSH terms

  • Adult
  • Aged
  • Alkaline Phosphatase / blood
  • Bilirubin / blood
  • Female
  • Hematopoiesis, Extramedullary
  • Hematopoietic Stem Cells / pathology
  • Humans
  • Kupffer Cells / pathology
  • Liver / pathology
  • Liver Diseases / blood
  • Liver Diseases / etiology*
  • Liver Diseases / pathology
  • Male
  • Megakaryocytes / pathology
  • Middle Aged
  • Primary Myelofibrosis / surgery*
  • Splenectomy / adverse effects*
  • gamma-Glutamyltransferase / blood

Substances

  • gamma-Glutamyltransferase
  • Alkaline Phosphatase
  • Bilirubin