The role of oral mucositis on the systemic inflammation parameter IL-8 in febrile neutropenic cancer patients

Cancer Invest. 2006 Aug-Sep;24(5):479-83. doi: 10.1080/07357900600814698.

Abstract

Purpose: Cancer patients treated with cytostatic drugs often develop oral mucositis, considered to be a mucosal injury in which various cytokines, such as interleukin 8 (IL-8), may play a role. Plasma IL-8 is a systemic inflammatory response parameter. This study investigated whether oral mucositis affects plasma IL-8 levels in febrile neutropenic cancer patients.

Patients and methods: Patients (n = 57) who were hospitalized with chemotherapy-induced neutropenic fever were scored for oral mucositis on the second day of hospitalization according to a validated oral mucositis assessment scale (OMAS) and WHO toxicity grading. Patients (n = 20) with a clinical sepsis or local bacterial infection were excluded from this evaluation. The remaining 37 patients were divided in groups with and without oral mucositis.

Results: The difference in plasma IL-8 level between patients with and without mucositis was not significant (P = 0.7). Similarly no difference was observed in the degree and duration of granulocytopenia.

Conclusion: These results indicate that low-grade oral mucositis is not related to the systemic plasma IL-8 level in febrile neutropenic cancer patients without a clinical sepsis or local bacterial infection.

Publication types

  • Comparative Study

MeSH terms

  • Adolescent
  • Adult
  • Antineoplastic Agents / adverse effects*
  • Child
  • Child, Preschool
  • Female
  • Fever / blood*
  • Fever / chemically induced
  • Humans
  • Inflammation Mediators / blood*
  • Interleukin-8 / blood*
  • Male
  • Middle Aged
  • Neoplasms / drug therapy
  • Neoplasms / immunology
  • Neutropenia / blood*
  • Neutropenia / chemically induced
  • Severity of Illness Index
  • Stomatitis / blood*
  • Stomatitis / chemically induced
  • Stomatitis / pathology

Substances

  • Antineoplastic Agents
  • CXCL8 protein, human
  • Inflammation Mediators
  • Interleukin-8