Cutting Edge: Allele-specific and peptide-dependent interactions between KIR3DL1 and HLA-A and HLA-B

J Immunol. 2007 Jan 1;178(1):33-7. doi: 10.4049/jimmunol.178.1.33.

Abstract

Although it is clear that KIR3DL1 recognizes Bw4(+) HLA-B, the role of Bw4(+) HLA-A allotypes as KIR3DL1 ligands is controversial. We therefore examined the binding of tetrameric HLA-A and -B complexes, including HLA*2402, a common Bw4(+) HLA-A allotype, to KIR3DL1*001, *005, *007, and *1502 allotypes. Only Bw4(+) tetramers bound KIR3DL1. Three of four HLA-A*2402 tetramers bound one or more KIR3DL1 allotypes and all four KIR3DL1 allotypes bound to one or more HLA-A*2402 tetramers, but with different binding specificities. Only KIR3DL1*005 bound both HLA-A*2402 and HLA-B*5703 tetramers. HLA-A*2402-expressing target cells were resistant to lysis by NK cells expressing KIR3DL1*001 or *005. This study shows that HLA-A*2402 is a ligand for KIR3DL1 and demonstrates how the binding of KIR3DL1 to Bw4(+) ligands depends upon the bound peptide as well as HLA and KIR3DL1 polymorphism.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alleles
  • Amino Acid Sequence
  • HLA-A Antigens / genetics
  • HLA-A Antigens / metabolism*
  • HLA-A24 Antigen
  • HLA-B Antigens / genetics
  • HLA-B Antigens / metabolism*
  • Histocompatibility Antigens Class I / genetics
  • Histocompatibility Antigens Class I / metabolism
  • Humans
  • Killer Cells, Natural / immunology*
  • Ligands
  • Models, Molecular
  • Molecular Sequence Data
  • Peptides / chemistry
  • Polymorphism, Genetic*
  • Receptors, Immunologic / genetics
  • Receptors, Immunologic / metabolism*
  • Receptors, KIR
  • Receptors, KIR3DL1

Substances

  • HLA-A Antigens
  • HLA-A*24:02 antigen
  • HLA-A24 Antigen
  • HLA-B Antigens
  • Histocompatibility Antigens Class I
  • KIR3DL1 protein, human
  • Ligands
  • Peptides
  • Receptors, Immunologic
  • Receptors, KIR
  • Receptors, KIR3DL1