MS-8209, a new Amphotericin B derivative that inhibits HIV-1 replication in vitro and restores T-cell activation via the CD3/TcR in HIV-infected CD4+ cells

AIDS. 1991 Dec;5(12):1453-61. doi: 10.1097/00002030-199112000-00007.

Abstract

A new Amphotericin B derivative, MS-8209, which retains high antifungal activity with greatly reduced toxicity and improved solubility, has been developed. We investigated the antiviral properties of MS-8209 in Jurkat and CEM T-cell lines and in peripheral blood mononuclear cells infected in vitro with HIV-1BRU. Our results demonstrate, by determination of reverse transcriptase activity and p24 antigen level titration in cell culture supernatants, that MS-8209 inhibits HIV-1 replication in all cell types at concentrations without cytotoxicity. MS-8209 also prevents membrane expression of the HIV-1 large envelope glycoprotein gp120 and the decrease in CD4 level at the surface of infected cells. HIV-1-infected Jurkat cells exhibit a severe signalling defect at CD3 stimulation. Treatment with MS-8209 restores normal responsiveness at CD3 as assessed by measurement of inositol triphosphate accumulation and calcium flux. Finally, our results indicate that MS-8209 inhibits HIV-1BRU replication without preventing virus binding and penetration into target cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amphotericin B / analogs & derivatives*
  • Amphotericin B / pharmacology
  • Antigens, Differentiation, T-Lymphocyte / physiology
  • Antiviral Agents / pharmacology*
  • CD3 Complex
  • CD4-Positive T-Lymphocytes / drug effects
  • CD4-Positive T-Lymphocytes / immunology
  • CD4-Positive T-Lymphocytes / microbiology*
  • Cell Line
  • HIV Core Protein p24 / analysis
  • HIV Envelope Protein gp120 / metabolism
  • HIV Reverse Transcriptase
  • HIV-1 / drug effects*
  • Humans
  • Kinetics
  • Lymphocyte Activation / drug effects*
  • Microscopy, Fluorescence
  • RNA-Directed DNA Polymerase / metabolism
  • Receptors, Antigen, T-Cell / physiology
  • Type C Phospholipases / metabolism
  • Virus Replication / drug effects*

Substances

  • Antigens, Differentiation, T-Lymphocyte
  • Antiviral Agents
  • CD3 Complex
  • HIV Core Protein p24
  • HIV Envelope Protein gp120
  • Receptors, Antigen, T-Cell
  • MS 8209
  • Amphotericin B
  • HIV Reverse Transcriptase
  • RNA-Directed DNA Polymerase
  • Type C Phospholipases