Terminal differentiation of lymphocytes depends on Blimp-1

Curr Opin Immunol. 2007 Apr;19(2):156-62. doi: 10.1016/j.coi.2007.01.003. Epub 2007 Feb 8.

Abstract

B lymphocyte induced maturation protein 1 (Blimp-1) has long been considered a master regulator of the terminal differentiation of B cells into antibody-secreting plasma cells. Gene-targeting experiments have now demonstrated that quantitative changes in Blimp-1 expression define plasma cell ontogeny--a process that requires the continual function of Blimp-1. Recently, new roles for Blimp-1 have been revealed, as a suppressor of diffuse large B cell lymphoma and as a key regulator of T-cell differentiation. Blimp-1 is expressed in differentiated effector T cells and controls their homeostasis. These new findings suggest that Blimp-1 has a conserved function in the final differentiation of both the cellular and the humoral arm of the adaptive immune response.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Cell Differentiation / genetics
  • Homeostasis / genetics
  • Homeostasis / immunology
  • Humans
  • Lymphoma, B-Cell / genetics*
  • Lymphopoiesis / genetics*
  • Mice
  • Plasma Cells / immunology
  • Positive Regulatory Domain I-Binding Factor 1
  • Repressor Proteins / analysis
  • Repressor Proteins / genetics
  • Repressor Proteins / physiology*
  • T-Lymphocytes / immunology*
  • Transcription Factors / analysis
  • Transcription Factors / genetics
  • Transcription Factors / physiology*

Substances

  • Prdm1 protein, mouse
  • Repressor Proteins
  • Transcription Factors
  • Positive Regulatory Domain I-Binding Factor 1