Protein/DNA interactions involving ATF/AP1-, CCAAT-, and HiNF-D-related factors in the human H3-ST519 histone promoter: cross-competition with transcription regulatory sites in cell cycle controlled H4 and H1 histone genes

J Cell Biochem. 1991 Dec;47(4):337-51. doi: 10.1002/jcb.240470408.

Abstract

Protein/DNA interactions of the H3-ST519 histone gene promoter were analyzed in vitro. Using several assays for sequence specificity, we established binding sites for ATF/AP1-, CCAAT-, and HiNF-D related DNA binding proteins. These binding sites correlate with two genomic protein/DNA interaction domains previously established for this gene. We show that each of these protein/DNA interactions has a counterpart in other histone genes: H3-ST519 and H4-F0108 histone genes interact with ATF- and HiNF-D related binding activities, whereas H3-ST519 and H1-FNC16 histone genes interact with the same CCAAT-box binding activity. These factors may function in regulatory coupling of the expression of different histone gene classes. We discuss these results within the context of established and putative protein/DNA interaction sites in mammalian histone genes. This model suggests that heterogeneous permutations of protein/DNA interaction elements, which involve both general and cell cycle regulated DNA binding proteins, may govern the cellular competency to express and coordinately control multiple distinct histone genes.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Activating Transcription Factors
  • Base Sequence
  • Binding Sites / genetics
  • Binding, Competitive
  • Blood Proteins / metabolism*
  • CCAAT-Enhancer-Binding Proteins
  • Cell Cycle / physiology*
  • DNA / metabolism*
  • DNA Fingerprinting
  • DNA Probes
  • DNA-Binding Proteins / metabolism*
  • Deoxyribonuclease I / metabolism
  • Gene Expression Regulation*
  • Histones / genetics*
  • Histones / metabolism
  • Humans
  • Methylation
  • Molecular Sequence Data
  • Promoter Regions, Genetic*
  • Sulfuric Acid Esters / metabolism
  • Transcription Factors / metabolism*
  • Transcription, Genetic

Substances

  • Activating Transcription Factors
  • Blood Proteins
  • CCAAT-Enhancer-Binding Proteins
  • DNA Probes
  • DNA-Binding Proteins
  • HINF-D protein, human
  • Histones
  • Sulfuric Acid Esters
  • Transcription Factors
  • DNA
  • Deoxyribonuclease I
  • dimethyl sulfate