Unstimulated amylase secretion is proteoglycan-dependent in rat parotid acinar cells

Arch Biochem Biophys. 2008 Jan 15;469(2):165-73. doi: 10.1016/j.abb.2007.10.008. Epub 2007 Oct 23.

Abstract

It is well-known that amylase is secreted in response to extracellular stimulation from the acinar cells. However, amylase is also secreted without stimulation. We distinguished vesicular amylase as a newly synthesized amylase from the accumulated amylase in secretory granules by short time pulse and chased with (35)S-amino acid. The newly synthesized amylase was secreted without stimulation from secretory vesicles in rat parotid acinar cells. The secretion process did not include microtubules, but was related to microfilaments. p-Nitrophenyl beta-xyloside, an inhibitor of proteoglycan synthesis, inhibited the newly synthesized amylase secretion. This indicated that the newly synthesized amylase was secreted from secretory vesicles, not via the constitutive-like secretory route, which includes the immature secretory granules, and that proteoglycan synthesis was required for secretory vesicle formation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amylases / metabolism*
  • Animals
  • Biological Transport
  • Brefeldin A / pharmacology
  • Cells, Cultured
  • Cytoskeleton / metabolism
  • Glycosides / pharmacology
  • Microscopy, Electron, Transmission
  • Microscopy, Fluorescence
  • Models, Biological
  • Parotid Gland / metabolism*
  • Proteoglycans / metabolism*
  • Rats

Substances

  • Glycosides
  • Proteoglycans
  • 4-nitrophenyl beta-D-xyloside
  • Brefeldin A
  • Amylases