Abstract
Assessment of 1177 human immunodeficiency virus (HIV) resistance genotypes at an HIV/AIDS clinic showed a decrease in the incidence of the K65R mutation, from 15.2% of isolates during the period 2002-2004 to 2.7% of isolates during the period 2005-2006 (P < .001), despite elevated and stable rates of tenofovir use. A reduction in the rate of coadministration of didanosine (from 41.6% of patients in 2004 to 0.8% of patients in 2006; P < .001) largely explained this observation.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adenine / analogs & derivatives
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Adenine / pharmacology
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Anti-HIV Agents / pharmacology*
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Didanosine / pharmacology
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Drug Resistance, Multiple, Viral / genetics*
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HIV Infections / metabolism*
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HIV Reverse Transcriptase / antagonists & inhibitors
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HIV Reverse Transcriptase / genetics*
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HIV-1 / drug effects
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HIV-1 / enzymology
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HIV-1 / genetics
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Humans
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Mutation
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Organophosphonates / pharmacology
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Retrospective Studies
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Reverse Transcriptase Inhibitors / pharmacology*
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Tenofovir
Substances
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Anti-HIV Agents
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Organophosphonates
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Reverse Transcriptase Inhibitors
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Tenofovir
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HIV Reverse Transcriptase
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Adenine
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Didanosine