cAMP modulates macrophage development by suppressing M-CSF-induced MAPKs activation

Cell Mol Immunol. 2008 Apr;5(2):153-7. doi: 10.1038/cmi.2008.19.

Abstract

M-CSF is a key cytokine in macrophage development by inducing MAPKs activation, and cAMP can inhibit MAPKs activation induced by inflammatory stimuli. To explore the effects of cAMP on M-CSF-induced MAPKs activation and on macrophage development, the model of bone marrow-derived murine macrophages (BMMs) was used. The effects of cAMP on M-CSF-induced MAPKs activation were analyzed by Western blotting assay, and the effects of cAMP on CD14 and F4/80 expression during macrophage development were examined by FACS analysis. Macrophage morphology showed the successful establishment of the model of macrophage development. Western blotting assay revealed that M-CSF activated ERK, JNK and p38 in both mature and immature macrophages, and cAMP inhibited M-CSF-induced ERK, JNK and p38 activation in a time-dependent manner. FACS analysis revealed that macrophage development was impaired with cAMP pretreatment. In conclusion, cAMP modulates macrophage development by suppressing M-CSF-induced MAPKs activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, Differentiation / genetics
  • Antigens, Differentiation / metabolism
  • Blotting, Western
  • Cell Differentiation / drug effects*
  • Cells, Cultured
  • Cyclic AMP / metabolism
  • Cyclic AMP / pharmacology*
  • Enzyme Activation / drug effects
  • Female
  • Gene Expression Regulation / drug effects
  • Lipopolysaccharide Receptors / genetics
  • Lipopolysaccharide Receptors / metabolism
  • Macrophage Colony-Stimulating Factor / metabolism*
  • Macrophages / cytology*
  • Macrophages / drug effects*
  • Macrophages / enzymology
  • Macrophages / immunology
  • Mice
  • Mice, Inbred C57BL
  • Mitogen-Activated Protein Kinases / metabolism*
  • Time Factors

Substances

  • Antigens, Differentiation
  • Lipopolysaccharide Receptors
  • monocyte-macrophage differentiation antigen
  • Macrophage Colony-Stimulating Factor
  • Cyclic AMP
  • Mitogen-Activated Protein Kinases