A nonsense mutation (E1978X) in the ATM gene is associated with breast cancer

Breast Cancer Res Treat. 2009 Nov;118(1):207-11. doi: 10.1007/s10549-008-0189-9. Epub 2008 Sep 21.

Abstract

Blood relatives of patients with ataxia-telangiectasia (A-T) have an increased risk to develop breast cancer. Allelic heterogeneity has made it difficult to confirm the role of ATM, the gene mutated in A-T, for breast cancer susceptibility in the general population. We now report that a nonsense mutation, p.E1978X (c.5932G>T), is both a classical A-T mutation and a breast cancer susceptibility allele in Eastern European populations. In a case-control study from Belarus, the E1978X mutation was identified in 10/1,891 Byelorussian breast cancer cases (0.5%) compared with 1/1,019 population controls [odds ratio (OR): 5.4; 95% confidence interval (95% CI), 0.7-42.4, P = 0.1]. A second case-control study from Russia identified the E1978X mutation in two Russian and one Ukrainian cases out of 611 breast cancer patients but not in any Russian or Ukrainian controls (P = 0.1). In a third case-control study from Poland, E1978X was observed in 7/3,910 Polish breast cancer cases (0.2%) compared with 1/2,010 cancer-free population controls (OR: 3.6; 95% CI: 0.4-29.3, P = 0.4). In the combined analysis, E1978X was significantly associated with breast cancer (Mantel-Haenszel OR: 5.6, 95% CI: 1.3-21.4, P = 0.01). Taken together, this study provides first evidence for the association of a common A-T causing mutation with breast cancer in Eastern European founder populations.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Ataxia Telangiectasia Mutated Proteins
  • Breast Neoplasms / epidemiology
  • Breast Neoplasms / ethnology
  • Breast Neoplasms / genetics*
  • Carcinoma, Ductal, Breast / epidemiology
  • Carcinoma, Ductal, Breast / ethnology
  • Carcinoma, Ductal, Breast / genetics*
  • Carcinoma, Lobular / epidemiology
  • Carcinoma, Lobular / ethnology
  • Carcinoma, Lobular / genetics*
  • Case-Control Studies
  • Cell Cycle Proteins / genetics*
  • Cell Cycle Proteins / physiology
  • Codon, Nonsense*
  • DNA-Binding Proteins / genetics*
  • DNA-Binding Proteins / physiology
  • Family Health
  • Female
  • Founder Effect
  • Gene Frequency
  • Genetic Predisposition to Disease
  • Genotype
  • Humans
  • Middle Aged
  • Odds Ratio
  • Point Mutation*
  • Poland / epidemiology
  • Protein Serine-Threonine Kinases / genetics*
  • Protein Serine-Threonine Kinases / physiology
  • Republic of Belarus / epidemiology
  • Russia / epidemiology
  • Sequence Analysis, DNA
  • Tumor Suppressor Proteins / genetics*
  • Tumor Suppressor Proteins / physiology
  • Ukraine / epidemiology
  • Ukraine / ethnology

Substances

  • Cell Cycle Proteins
  • Codon, Nonsense
  • DNA-Binding Proteins
  • Tumor Suppressor Proteins
  • ATM protein, human
  • Ataxia Telangiectasia Mutated Proteins
  • Protein Serine-Threonine Kinases