Transcriptome analysis of the zebrafish mind bomb mutant

Mol Genet Genomics. 2009 Jan;281(1):77-85. doi: 10.1007/s00438-008-0395-5. Epub 2008 Nov 13.

Abstract

Mind bomb (Mib) facilitates Notch signaling pathway by promoting the endocytosis of Notch ligand. The zebrafish mib ( ta52b ) mutant has a defect in its ubiquitin ligase activity which is necessary to inhibit the neurogenesis, resulting in a neuronal hyperplasia. Several genes regulated in the mib ( ta52b ) mutant have been well established, however, there were relatively few reports about the transcriptome profile. To identify the genes differentially expressed in the mib ( ta52b ) mutant, genome-wide analysis was performed using serial analysis of gene expression. Three hundred and thirty-five transcripts were identified whose expressions were significantly altered in the mib ( ta52b ) mutant as compared with the wild-type. Interestingly, it was suggested that the mib ( ta52b ) mutation may affect not only neurogenesis but also mesoderm development. These results provide new insights into Notch signaling pathway.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • DNA Primers / genetics
  • Gene Expression Profiling
  • Gene Expression Regulation, Developmental
  • Genome-Wide Association Study
  • In Situ Hybridization
  • Mesoderm / embryology
  • Mutation
  • Neurogenesis / genetics
  • Oligonucleotide Array Sequence Analysis
  • Receptors, Notch / metabolism
  • Signal Transduction
  • Ubiquitin-Protein Ligases / genetics*
  • Zebrafish / embryology
  • Zebrafish / genetics*
  • Zebrafish / metabolism
  • Zebrafish Proteins / genetics*
  • Zebrafish Proteins / metabolism

Substances

  • DNA Primers
  • Receptors, Notch
  • Zebrafish Proteins
  • Ubiquitin-Protein Ligases
  • mib1 protein, zebrafish