Mutations in C2orf37, encoding a nucleolar protein, cause hypogonadism, alopecia, diabetes mellitus, mental retardation, and extrapyramidal syndrome

Am J Hum Genet. 2008 Dec;83(6):684-91. doi: 10.1016/j.ajhg.2008.10.018. Epub 2008 Nov 20.

Abstract

Hypogonadism, alopecia, diabetes mellitus, mental retardation, and extrapyramidal syndrome (also referenced as Woodhouse-Sakati syndrome) is a rare autosomal recessive multisystemic disorder. We have identified a founder mutation consisting of a single base-pair deletion in C2orf37 in eight families of Saudi origin. Three other loss-of-function mutations were subsequently discovered in patients of different ethnicities. The gene encodes a nucleolar protein of unknown function, and the cellular phenotype observed in patient lymphoblasts implicates a role for the nucleolus in the pathogenesis of this disease. Our findings expand the list of human disorders linked to the nucleolus and further highlight the developmental and/or maintenance functions of this organelle.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alopecia / genetics
  • Amino Acid Sequence
  • Basal Ganglia Diseases / genetics
  • Base Sequence / genetics
  • Chromosomes, Human, Pair 2*
  • Conserved Sequence
  • Diabetes Mellitus / genetics
  • Female
  • Genes, Recessive
  • Genetic Linkage
  • Genome, Human
  • Haplotypes
  • Homozygote
  • Humans
  • Hypogonadism / genetics
  • Intellectual Disability / genetics
  • Lod Score
  • Male
  • Molecular Sequence Data
  • Mutation*
  • Nuclear Proteins / chemistry
  • Nuclear Proteins / genetics*
  • Nuclear Proteins / metabolism
  • Open Reading Frames*
  • Pedigree
  • Physical Chromosome Mapping
  • Sequence Analysis, DNA
  • Sequence Deletion
  • Syndrome
  • Ubiquitin-Protein Ligase Complexes

Substances

  • DCAF17 protein, human
  • Nuclear Proteins
  • Ubiquitin-Protein Ligase Complexes