Clinical and immunohistochemical features associated with a response to bortezomib in patients with multiple myeloma

Clin Cancer Res. 2009 Jan 15;15(2):714-22. doi: 10.1158/1078-0432.CCR-08-1022.

Abstract

Purpose: Multiple myeloma is an incurable disease with heterogeneous clinical behavior. Bortezomib has offered some patients with relapsed and refractory disease an opportunity for prolonged survival. However, there remains a paucity of data in patients treated with bortezomib that accurately delineates and identifies such patients. This information is crucial to guide management.

Experimental design: In this study, we aimed to identify the patients most likely to respond to bortezomib salvage therapy. We analyzed the baseline clinical variables and profiled the baseline expression of a broad range of immunohistochemical markers of cell cycle activity, apoptosis, and angiogenesis in a large cohort of multiply relapsed myeloma patients recruited to one of two prospective multicentre trials assessing the efficacy of bortezomib salvage therapy.

Results: Using the European Group for Bone Marrow Transplantation criteria, response (complete or partial) to bortezomib salvage therapy was associated with a previous history of complete response to alternative antimyeloma treatment. Patients who expressed cyclin D1 were more likely to achieve a response. In contrast, patients who expressed p16(INK4A), cytoplasmic p53, and the highest intensity of Bcl-2 staining had a poor response. Patients who achieved a response to bortezomib and those patients who expressed cyclin D1 at baseline showed a significant survival advantage. Patients who expressed FGFR3, a poor prognostic marker, responded equally well and had similar outcomes with bortezomib compared with FGFR3-negative patients.

Conclusions: Baseline clinical variables and selective immunohistochemical markers expressed by patients may be used effectively to identify patients that are most likely to achieve a meaningful clinical response to bortezomib salvage therapy.

Publication types

  • Multicenter Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Antineoplastic Agents / therapeutic use*
  • Boronic Acids / therapeutic use*
  • Bortezomib
  • Cell Cycle
  • Cohort Studies
  • Cyclin D1 / biosynthesis
  • Cyclin-Dependent Kinase Inhibitor p16 / biosynthesis
  • Female
  • Humans
  • Immunohistochemistry / methods*
  • Male
  • Middle Aged
  • Multiple Myeloma / drug therapy*
  • Prognosis
  • Proto-Oncogene Proteins c-bcl-2 / biosynthesis
  • Pyrazines / therapeutic use*
  • Treatment Outcome
  • Tumor Suppressor Protein p53 / biosynthesis

Substances

  • Antineoplastic Agents
  • Boronic Acids
  • Cyclin-Dependent Kinase Inhibitor p16
  • Proto-Oncogene Proteins c-bcl-2
  • Pyrazines
  • TP53 protein, human
  • Tumor Suppressor Protein p53
  • Cyclin D1
  • Bortezomib