A ribosomal protein L23-nucleophosmin circuit coordinates Mizl function with cell growth

Nat Cell Biol. 2008 Sep;10(9):1051-61. doi: 10.1038/ncb1764.

Abstract

The Myc-associated zinc-finger protein, Miz1, is a negative regulator of cell proliferation and induces expression of the cell-cycle inhibitors p15(Ink4b) and p21(Cip1). Here we identify the ribosomal protein L23 as a negative regulator of Miz1-dependent transactivation. L23 exerts this function by retaining nucleophosmin, an essential co-activator of Miz1 required for Miz1-induced cell-cycle arrest, in the nucleolus. Mutant forms of nucleophosmin found in acute myeloid leukaemia fail to co-activate Miz1 and re-localize it to the cytosol. As L23 is encoded by a direct target gene of Myc, this regulatory circuit may provide a feedback mechanism that links translation of Myc target genes and cell growth to Miz1-dependent cell-cycle arrest.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alleles
  • Animals
  • Cell Proliferation
  • Feedback, Physiological
  • HeLa Cells
  • Humans
  • Models, Biological
  • Mutant Proteins / metabolism
  • Nuclear Proteins / metabolism*
  • Nucleophosmin
  • Protein Inhibitors of Activated STAT / antagonists & inhibitors
  • Protein Inhibitors of Activated STAT / chemistry
  • Protein Inhibitors of Activated STAT / metabolism*
  • Protein Structure, Tertiary
  • Proto-Oncogene Proteins c-myc / metabolism
  • Rats
  • Ribosomal Proteins / metabolism*

Substances

  • Mutant Proteins
  • NPM1 protein, human
  • Nuclear Proteins
  • PIAS2 protein, human
  • Protein Inhibitors of Activated STAT
  • Proto-Oncogene Proteins c-myc
  • Ribosomal Proteins
  • ribosomal protein L17
  • Nucleophosmin