Inhibition of human immunodeficiency virus type 1-induced cell fusion by recombinant human interferons

J Virol. 1991 Nov;65(11):6325-30. doi: 10.1128/JVI.65.11.6325-6330.1991.

Abstract

Pretreatment of HeLa T4 cells with recombinant alpha, beta, or gamma interferon (IFN) was found to significantly inhibit syncytium formation induced by the human immunodeficiency virus type 1 (HIV-1) envelope glycoprotein. All three IFNs were found to be potent inhibitors of fusion in a system in which Spodoptera frugiperda cells, infected with a baculovirus recombinant expressing the HIV-1 envelope protein, were cocultivated with HeLa T4 cells. In addition, these IFNs were also found to block HeLa T4 cell fusion induced by the HIV-1 envelope proteins expressed from a vaccinia virus recombinant. Furthermore, the IFNs inhibited cell fusion between HIV-1 envelope glycoprotein-expressing cells and either immortalized or fresh CD4+ lymphocytes pretreated with the IFNs. These results suggest that further testing of human IFNs for therapy of HIV-1 infection will be of interest.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Baculoviridae / genetics
  • Cell Fusion / drug effects*
  • Cell Line
  • Dose-Response Relationship, Drug
  • Giant Cells / cytology*
  • Giant Cells / drug effects
  • HIV-1 / drug effects
  • HIV-1 / physiology*
  • HeLa Cells
  • Humans
  • Interferon Type I / pharmacology*
  • Interferon-beta / pharmacology*
  • Interferon-gamma / pharmacology*
  • Moths
  • Recombinant Proteins / pharmacology
  • Transfection
  • Vaccinia virus / genetics
  • Viral Envelope Proteins / genetics

Substances

  • Interferon Type I
  • Recombinant Proteins
  • Viral Envelope Proteins
  • Interferon-beta
  • Interferon-gamma