Relationship between non-fibrillary amyloid precursors and cell processes in the cortical neuropil of Alzheimer patients

Neurosci Lett. 1991 Aug 5;129(1):119-22. doi: 10.1016/0304-3940(91)90734-b.

Abstract

We examined the ultrastructural localization of amyloid beta-protein in 8 Alzheimer neocortical biopsies. Intense immunoreactivity was located extracellularly on amyloid fibrils and amorphous material. Amorphous labelled material was also found in cell processes. No ultrastructural cell marker, such as glial fibrils, glycogen, tubules, paired helical filaments (PFHs) or synaptic vesicles could be seen in these processes that could allow their identification as glial processes, neurites or presynaptic terminals, respectively; occasional membrane stacks were observed. These findings suggest that preamyloid deposits are related to cell processes and, by elimination, that postsynaptic terminals may be involved in abnormal metabolism of the amyloid fibril precursors.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Aged
  • Alzheimer Disease / metabolism*
  • Alzheimer Disease / pathology
  • Amyloid beta-Protein Precursor / analysis*
  • Amyloid beta-Protein Precursor / immunology
  • Cerebral Cortex / chemistry*
  • Cerebral Cortex / ultrastructure
  • Humans
  • Microscopy, Immunoelectron
  • Middle Aged
  • Neurofibrillary Tangles / chemistry*

Substances

  • Amyloid beta-Protein Precursor