Moderately decreased cholesterol absorption rates are associated with a large atheroprotective effect

Arterioscler Thromb Vasc Biol. 2009 Nov;29(11):1745-50. doi: 10.1161/ATVBAHA.109.194605. Epub 2009 Aug 6.

Abstract

Objective: Human treatment with ezetimibe results in a moderate 50% to 54% decrease in cholesterol absorption and a 15% to 20% decrease in plasma LDL-cholesterol levels; nevertheless, the efficacy of ezetimibe therapy has been recently challenged by the ENHANCE trial. We examined the efficacy of a moderate decrease in cholesterol absorption in preventing atherosclerosis formation in the mouse.

Methods and results: Congenic 14DKK animals, consisting of a castaneus (CASA/Rk) chromosome 14 interval introgressed onto the C57BL/6J background, displayed a moderate decrease in cholesterol absorption rates. The effect of moderately decreased absorption on atherosclerosis formation was determined in 14DKK apolipoprotein E knockouts (14DKK-apoEKO). When compared to chow diet-fed control apoEKO mice, congenic 14DKK-apoEKO displayed a moderate 41% decrease in cholesterol absorption rates, 30% to 37% decrease in plasma cholesterol levels, and a 70% decrease in atherosclerosis formation. Studies on cholesterol efflux and reverse cholesterol transport (RCT) from 14DKK bone marrow-derived macrophages rejected a 14DKK interval-dependent atheroprotective effects that operate in macrophages. In contrast, 14DKK-apoEKO congenics were characterized by a 60% increase in RCT from peripheral tissue macrophages.

Conclusions: These studies strongly suggest that moderately decreased cholesterol absorption rates result in a large atheroprotective effect attributable to a decrease in plasma cholesterol levels and an increase in RCT from peripheral tissue macrophages.

Publication types

  • Comparative Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Analysis of Variance
  • Animals
  • Animals, Congenic
  • Anticholesteremic Agents / pharmacology*
  • Apolipoproteins E / metabolism
  • Apolipoproteins E / pharmacology
  • Atherosclerosis / metabolism*
  • Atherosclerosis / prevention & control*
  • Biological Transport / drug effects
  • Biological Transport / genetics
  • Cholesterol, LDL / drug effects*
  • Cholesterol, LDL / genetics
  • Cholesterol, LDL / metabolism*
  • Disease Models, Animal
  • Lipid Metabolism / drug effects
  • Macrophages / drug effects
  • Macrophages / metabolism
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Probability
  • Random Allocation
  • Statistics, Nonparametric

Substances

  • Anticholesteremic Agents
  • Apolipoproteins E
  • Cholesterol, LDL