Prevention of UV-induced damage to the anterior segment using class I UV-absorbing hydrogel contact lenses

Invest Ophthalmol Vis Sci. 2010 Jan;51(1):172-8. doi: 10.1167/iovs.09-3996. Epub 2009 Aug 26.

Abstract

Purpose: To determine whether class I ultraviolet (UV) light-blocking contact lenses prevent UV-induced pathologic changes in a rabbit model.

Methods: Twelve rabbits were assigned to 1 of 3 treatment groups (n = 4), as follows: senofilcon A (class I UV blocking) contact lenses; lotrafilcon A contact lenses (no reported UV blocking); no contact lens. The contralateral eye was patched without a contact lens. Animals received UV-B (1.667 J/cm(2)) exposure daily for 5 days. Postmortem tissue was examined as follows: in the cornea, the expression of matrix-metalloproteinases (MMPs) was evaluated by zymography, and apoptosis was evaluated by TUNEL and caspase-3 ELISA; ascorbate in the aqueous humor was evaluated by nuclear magnetic resonance spectroscopy; crystalline lens apoptosis was evaluated by TUNEL and caspase-3 ELISA.

Results: Exposed corneas showed a significant increase in MMP-2 and -9, TUNEL-positive cells, and caspase-3 activity in the lotrafilcon A group compared with the senofilcon A group (all P = 0.03). A significant decrease in aqueous humor ascorbate was observed in the exposed lotrafilcon A lens-wearing group compared with the exposed senofilcon A lens-wearing group (P = 0.03). Exposed crystalline lenses had significantly increased caspase-3 activity in the lotrafilcon A group compared with the senofilcon A group (P = 0.03). Increased numbers of TUNEL-positive cells were noted in both the lotrafilcon A and the non-contact lens groups.

Conclusions: The authors show that senofilcon A class I UV-blocking contact lenses are capable of protecting the cornea, aqueous humor, and crystalline lens of rabbits from UV-induced pathologic changes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Anterior Eye Segment / metabolism
  • Anterior Eye Segment / radiation effects*
  • Apoptosis
  • Aqueous Humor / metabolism
  • Aqueous Humor / radiation effects
  • Ascorbic Acid / metabolism
  • Caspase 3 / metabolism
  • Contact Lenses, Hydrophilic*
  • Cornea / metabolism
  • Cornea / pathology
  • Cornea / radiation effects
  • Enzyme-Linked Immunosorbent Assay
  • Eye Diseases / etiology
  • Eye Diseases / metabolism
  • Eye Diseases / prevention & control*
  • Hydrogels*
  • In Situ Nick-End Labeling
  • Lens, Crystalline / metabolism
  • Lens, Crystalline / pathology
  • Lens, Crystalline / radiation effects
  • Magnetic Resonance Spectroscopy
  • Matrix Metalloproteinases / metabolism
  • Prospective Studies
  • Rabbits
  • Radiation Injuries, Experimental / etiology
  • Radiation Injuries, Experimental / metabolism
  • Radiation Injuries, Experimental / prevention & control*
  • Radiation Protection / methods
  • Silicones*
  • Ultraviolet Rays / adverse effects*

Substances

  • Hydrogels
  • Silicones
  • lotrafilcon A
  • senofilcon A
  • Caspase 3
  • Matrix Metalloproteinases
  • Ascorbic Acid