Altered protein-expressing profile in hPNAS4-induced apoptosis in A549 human lung adenocarcinoma cells

J Cell Biochem. 2009 Dec 1;108(5):1211-9. doi: 10.1002/jcb.22353.

Abstract

Human PNAS4 (hPNAS4) is a recently identified pro-apoptosis gene, which is able to induce apoptosis in A549 human lung adenocarcinoma cells following its overexpression. In this work, we investigated the changes of protein profile in hPNAS4-induced apoptosis in A549 cells through proteomic strategy consisting of two-dimensional electrophoresis (2-DE) coupled with MALDI-Q-TOF mass spectrometry. A total of 20 different proteins with more than 3.0-fold change in expression, including 5 up-regulated and 15 down-regulated proteins were successfully identified by database search. The mRNA transcription levels of the different proteins were further examined by RT-PCT. Functional analyses showed these different proteins are involved in diverse biological processes including metabolism, proteolysis, signal transduction, apoptosis, and redox regulation. Two essential apoptosis-associated protein, annexin A1 and prothymosin alpha, were confirmed by Western blot and showed consistent changes with proteomic detection. Our data provide molecular evidence and possible associated pathway in hPNAS4-induced apoptosis through proteomic strategy, which should be contributed to further investigation on biological function of hPNAS4.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenocarcinoma / genetics
  • Adenocarcinoma / metabolism*
  • Adenocarcinoma / pathology
  • Annexin A1 / metabolism
  • Apoptosis / physiology*
  • Apoptosis Regulatory Proteins / genetics*
  • Apoptosis Regulatory Proteins / metabolism
  • Carbon-Nitrogen Lyases
  • Cell Line, Tumor
  • Down-Regulation
  • Gene Expression Profiling
  • Gene Expression Regulation, Neoplastic
  • Humans
  • Lung Neoplasms / genetics
  • Lung Neoplasms / metabolism*
  • Lung Neoplasms / pathology
  • Oxidation-Reduction
  • Protein Precursors / metabolism
  • Signal Transduction
  • Thymosin / analogs & derivatives
  • Thymosin / metabolism
  • Up-Regulation

Substances

  • Annexin A1
  • Apoptosis Regulatory Proteins
  • Protein Precursors
  • prothymosin alpha
  • Thymosin
  • Carbon-Nitrogen Lyases
  • DESI2 protein, human