Abstract
A library of 30 beta-lactams has been prepared from (3R,4R)-3-[(R)-1'-(tbutyldimethylsilyloxy)-ethyl]-4-acetoxy-2-azetidinone, and the corresponding deacetoxy derivative, by sequential N- and O-functionalizations with various omega-alkenoyl and omega-arylalkanoyl chains. All compounds were selective inhibitors of hFAAH versus hMGL, and IC(50) values in the nanomolar range (5-14 nM) were recorded for the best representatives. From time-dependent preincubation and rapid dilution studies, and from docking analyses in a homology model of the target enzyme, a reversible mechanism of inhibition of hFAAH is proposed.
Publication types
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Comparative Study
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Research Support, Non-U.S. Gov't
MeSH terms
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Amidohydrolases / antagonists & inhibitors*
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Amidohydrolases / chemistry
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Amidohydrolases / metabolism
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Carbapenems / chemical synthesis
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Carbapenems / chemistry*
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Carbapenems / metabolism
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Carbapenems / pharmacology*
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Enzyme Inhibitors / chemical synthesis
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Enzyme Inhibitors / chemistry*
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Enzyme Inhibitors / metabolism
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Enzyme Inhibitors / pharmacology*
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Humans
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Inhibitory Concentration 50
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Models, Molecular
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Molecular Conformation
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Monoacylglycerol Lipases / antagonists & inhibitors*
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Monoacylglycerol Lipases / chemistry
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Monoacylglycerol Lipases / metabolism
Substances
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Carbapenems
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Enzyme Inhibitors
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Monoacylglycerol Lipases
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Amidohydrolases
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fatty-acid amide hydrolase