Production and N-glycosylation of recombinant human cell adhesion molecule L1 from insect cells using the stable expression system. Effect of dimethyl sulfoxide

J Biotechnol. 2010 Jan 15;145(2):130-8. doi: 10.1016/j.jbiotec.2009.10.018. Epub 2009 Nov 14.

Abstract

L1 is a cell adhesion molecule that is heavily glycosylated and is essential for normal development of the central nervous system. In this work, we compare the N-glycosylation of the L1 mutant that consists of immunoglobulin domains 5 and 6 (L1/Ig5-6), expressed in insect Spodoptera frugiperda Sf9 and Trichoplusia ni Tn cells, using the stable expression system. L1/Ig5-6 levels of 30 and 8mgl(-1) were obtained from the two cell lines, respectively. The N-glycans were characterized by high-performance anion-exchange-chromatography with pulsed-amperometric-detection and matrix-assisted laser desorption/ionization time-of-flight mass spectrometry. The N-glycans from Sf9 cells were more homogeneous and consisted predominantly of the paucimannose-type structure Manalpha6(Manalpha3)Manbeta4GlcNAcbeta4(Fucalpha6)GlcNAc. On the other hand, the N-glycans from Tn cells were more heterogeneous and consisted of paucimannose-type structures with or without terminal N-acetylglucosamine. Allergenic proximal alpha3-linked fucose was only found in Tn cells. Dimethyl sulfoxide at 1.5% concentration has been found to increase the levels of L1/Ig5-6 and the L1 ectodomain in the Sf9 and Tn cells, without affecting cell viability nor protein integrity. Furthermore, the N-glycan composition of L1/Ig5-6 was not affected by dimethyl sulfoxide, with only a slight increase in the percentage of the minor high-mannose-type structures.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cells, Cultured
  • Cloning, Molecular / methods*
  • Dimethyl Sulfoxide / pharmacology*
  • Genetic Enhancement / methods
  • Glycosylation
  • Humans
  • Neural Cell Adhesion Molecule L1 / physiology*
  • Protein Engineering / methods*
  • Recombinant Proteins / metabolism
  • Spodoptera / cytology
  • Spodoptera / drug effects
  • Spodoptera / physiology*
  • Transfection / methods*

Substances

  • Neural Cell Adhesion Molecule L1
  • Recombinant Proteins
  • Dimethyl Sulfoxide