Macrophage colony-stimulating factor (CSF-1) gene expression in human T-lymphocyte clones

Blood. 1991 Feb 15;77(4):780-6.

Abstract

Macrophage colony stimulating factor (CSF-1) is one of several cytokines that control the differentiation, survival, and proliferation of monocytes and macrophages. A set of 11 human T-cell clones, chosen for their phenotypic diversity, were tested for their ability to express CSF-1 mRNA. After 5 hours of stimulation with phorbol myristate acetate (PMA) + calcium ionophore (Cal), all T-cell clones expressed a major 4-kb transcript, a less abundant 2-kb transcript, and several other minor species. This pattern of expression is typical for CSF-1 mRNAs. Furthermore, of the two alloreactive T-cell clones analyzed, only one showed a definitive message for CSF-1 on specific antigenic stimulation, but with delayed kinetics and less efficiency. Both conditions of stimulation induced the release of CSF-1 protein by T cells in the culture medium. Together, these findings demonstrate for the first time that normal T cells are able to produce CSF-1, previous reports being limited to two cases of tumoral cells of the T-cell lineage.

MeSH terms

  • Antigens / immunology
  • Calcimycin / pharmacology
  • Clone Cells
  • Gene Expression*
  • Graft Rejection
  • Graft vs Host Disease / pathology
  • Humans
  • Isoantigens / immunology
  • Macrophage Colony-Stimulating Factor / biosynthesis
  • Macrophage Colony-Stimulating Factor / genetics*
  • Nucleic Acid Hybridization
  • RNA, Messenger / metabolism
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism*
  • Tetradecanoylphorbol Acetate / pharmacology
  • Transcription, Genetic

Substances

  • Antigens
  • Isoantigens
  • RNA, Messenger
  • Calcimycin
  • Macrophage Colony-Stimulating Factor
  • Tetradecanoylphorbol Acetate