Role of interleukin 8 in uterine natural killer cell regulation of extravillous trophoblast cell invasion

Placenta. 2010 Jul;31(7):595-601. doi: 10.1016/j.placenta.2010.04.012. Epub 2010 May 18.

Abstract

Background: Extravillous trophoblast cell (EVT) invasion of maternal tissues is critical for successful pregnancy. Decidual factors, including uterine natural killer (uNK) and T cell derived cytokines play a role in regulating this process. Interleukin (IL) 8 has been implicated as a regulator of EVT invasion.

Hypothesis: uNK cell stimulation of EVT invasion is associated with IL-8 levels.

Methods: CD8+, total decidual and CD56(+) uNK cells (8-10 and 12-14 weeks gestational age) were cultured. IL-8 mRNA and protein levels were determined. IL-8 receptors (IL-8RA and IL-8RB) were localised in first trimester placental bed biopsies. The effect of IL-8 +/- IL-8 neutralising antibodies and CD8+ T cell or uNK cell supernatants +/- IL-8 neutralising antibodies on EVT invasion was assessed. EVT secreted levels of MMP-2, MMP-9, uPA, PAI-1 and PAI-2 were assessed by substrate zymography or Western Blot.

Results: High levels of IL-8 protein and mRNA were detected in all samples. IL-8RA and IL-8RB were expressed by EVT. Exogenous IL-8 stimulated EVT invasion in a paracrine manner. uNK cell supernatants, but not CD8+ cell supernatants, stimulated EVT invasion. IL-8 neutralising antibody partially abrogated this uNK cell stimulated invasion. IL-8 increased levels of secreted MMP-2, but did not alter any of the other proteases or protease inhibitors tested.

Conclusion: uNK cell stimulation of EVT invasion is partially mediated by IL-8. Unstimulated CD8+ T cells do not alter EVT invasion despite secreting similar levels of IL-8 as uNK cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • CD8-Positive T-Lymphocytes / immunology
  • Cell Movement / physiology
  • Female
  • Humans
  • Interleukin-8 / immunology
  • Interleukin-8 / physiology*
  • Killer Cells, Natural / immunology*
  • Matrix Metalloproteinase 2 / metabolism
  • Pregnancy / immunology*
  • Pregnancy Trimester, First / immunology
  • Receptors, Interleukin-8A / metabolism
  • Receptors, Interleukin-8B / metabolism
  • Trophoblasts / immunology*
  • Trophoblasts / physiology*
  • Uterus / immunology*

Substances

  • Interleukin-8
  • Receptors, Interleukin-8A
  • Receptors, Interleukin-8B
  • Matrix Metalloproteinase 2