Antitumor activity of satraplatin in cisplatin-resistant oral squamous cell carcinoma cells

Head Neck. 2011 Mar;33(3):309-17. doi: 10.1002/hed.21445.

Abstract

Background: The aim of the current study was to identify the antitumor activity of satraplatin in paired cisplatin (CDDP)-resistant oral squamous cell carcinoma (OSCC) cell line and its parental cell line.

Methods: CDDP-resistant (KB-R) cells and parental cells (KB) pair were used. Viability was assessed using the MTT and clonogenic assay. Real-time polymerase chain reaction (PCR), glutathione (GSH) assay, and flow cytometric analysis were used for further assessment.

Results: KB-R cells did not show cross-resistance to satraplatin. The expression status of almost all transporters was upregulated in the KB-R cells. There was no difference in the GSH levels between the KB and KB-R cells. Flow cytometric analysis indicated that with satraplatin the G2/M phase was arrested in the KB-R cells. KB-R cells contain enriched side population cells.

Conclusion: These data suggested that satraplatin has antitumor activity against the CDDP-resistant OSCC cells. The mechanism of cross-resistance to platinum agents seems to be multifactorial.

Publication types

  • Comparative Study

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Carcinoma, Squamous Cell / drug therapy*
  • Carcinoma, Squamous Cell / pathology
  • Cell Proliferation / drug effects*
  • Cisplatin / pharmacology*
  • Confidence Intervals
  • Drug Resistance, Neoplasm*
  • Flow Cytometry
  • Humans
  • Mouth Neoplasms / drug therapy*
  • Mouth Neoplasms / pathology
  • Organoplatinum Compounds / pharmacology*
  • RNA, Messenger / analysis
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sensitivity and Specificity
  • Statistics, Nonparametric
  • Tumor Cells, Cultured / drug effects

Substances

  • Antineoplastic Agents
  • Organoplatinum Compounds
  • RNA, Messenger
  • satraplatin
  • Cisplatin