Clevudine-induced viral response, associated with continued reduction of HBsAg titer, was durable after the withdrawal of therapy

J Gastroenterol. 2011 Mar;46(3):410-4. doi: 10.1007/s00535-010-0354-x. Epub 2010 Dec 23.

Abstract

Background: This study was conducted to evaluate the durability of clevudine-induced viral response after the withdrawal of treatment.

Methods: Patients who showed a complete response [alanine aminotransferase (ALT) normalization and hepatitis B virus (HBV) DNA <4,700 copies/mL for hepatitis B envelope antigen (HBeAg)-negative patients; ALT normalization, HBV DNA <4,700 copies/mL, and HBeAg seroconversion for HBeAg-positive patients] in the previous clevudine phase III trials were followed for an additional 96 weeks without any treatment for hepatitis B.

Results: Of the 63 patients in the study cohort, 73% and 35% of the patients had HBV DNA <141,500 and <4,700 copies/mL, respectively, and 75% of the patients had normal ALT at the end of follow-up. HBeAg seroconversion was maintained in 81% of the patients and hepatitis B surface antigen (HBsAg) loss occurred in 3 patients. Continued HBsAg titer decrease (-0.5 log IU/mL) was observed in the sustained viral responders, suggesting the reduction of covalently closed circular DNA in hepatocytes.

Conclusions: The clevudine-induced viral response was durable in the majority of patients for 2 years after the withdrawal of treatment.

Publication types

  • Multicenter Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alanine Transaminase / blood
  • Antiviral Agents / therapeutic use*
  • Arabinofuranosyluracil / analogs & derivatives*
  • Arabinofuranosyluracil / therapeutic use
  • DNA, Viral / blood
  • Follow-Up Studies
  • Hepatitis B / blood
  • Hepatitis B / drug therapy*
  • Hepatitis B / immunology
  • Hepatitis B / virology
  • Hepatitis B Surface Antigens / blood*
  • Hepatitis B e Antigens / blood
  • Hepatitis B e Antigens / immunology
  • Hepatitis B virus / immunology*
  • Humans
  • Viral Load

Substances

  • Antiviral Agents
  • DNA, Viral
  • Hepatitis B Surface Antigens
  • Hepatitis B e Antigens
  • Arabinofuranosyluracil
  • Alanine Transaminase
  • clevudine