Coreceptors and HIV-1 pathogenesis

Curr HIV/AIDS Rep. 2011 Mar;8(1):45-53. doi: 10.1007/s11904-010-0069-x.

Abstract

The major HIV-1 coreceptors, CCR5 and CXCR4, mediate virus entry into CD4+ cells and are therefore a critical component of the HIV-1 life cycle. Alterations in coreceptor preference as well as the efficiency and mechanism of interaction between HIV-1 and CCR5 and/or CXCR4 has a significant influence on viral tropism, progression of disease, and response to coreceptor antagonists. In addition, these alterations influence the susceptibility of CD4+ T-cell, monocyte, and dendritic cell subsets to infection and therefore, are important for several facets of HIV-1 pathogenesis including the establishment of latent reservoirs, trafficking, and transmission. This review highlights recent literature that has advanced our understanding of the role of coreceptors in HIV-1 pathogenesis.

Publication types

  • Review

MeSH terms

  • CD4-Positive T-Lymphocytes / immunology
  • CD4-Positive T-Lymphocytes / virology
  • Dendritic Cells / immunology
  • Dendritic Cells / virology
  • Disease Progression
  • Disease Reservoirs / virology
  • HIV Infections / immunology*
  • HIV-1 / pathogenicity*
  • Humans
  • Monocytes / immunology
  • Monocytes / virology
  • Receptors, CCR5 / metabolism*
  • Receptors, CXCR4 / metabolism*
  • Viral Tropism

Substances

  • Receptors, CCR5
  • Receptors, CXCR4