Nasal nitric oxide and nitric oxide synthase expression in primary ciliary dyskinesia

Eur Respir J. 2011 Mar;37(3):572-7. doi: 10.1183/09031936.00044310. Epub 2011 Jan 27.

Abstract

No study has evaluated the correlation between different expression of nitric oxide synthase (NOS) isoforms in nasal epithelial cells and nasal NO (nNO) level in primary ciliary dyskinesia (PCD). Gene expression of endothelial (NOS3) and inducible NOS (NOS2) and their correlation with nNO level, ciliary function and morphology were studied in patients with PCD or secondary ciliary dyskinesia (SCD). NOS3 gene polymorphisms were studied in blood leukocytes. A total of 212 subjects were studied (48 with PCD, 161 with SCD and three normal subjects). nNO level correlated with mean ciliary beat frequency (p = 0.044; r = 0.174). The lower the nNO level the higher was the percentage of immotile cilia (p<0.001; r = -0.375). A significant positive correlation between NOS2 gene expression and nNO levels was demonstrated in all children (p = 0.001; r = 0.428), and this correlation was confirmed in patients with PCD (p = 0.019; r = 0.484). NOS2 gene expression was lower in PCD than in SCD (p = 0.04). The NOS3 isoform correlated with missing central microtubules (p = 0.048; r = 0.447). nNO levels were higher in PCD subjects with the NOS3 thymidine 894 mutation, and this was associated with a higher ciliary beat frequency (p = 0.045). These results demonstrate a relationship between nNO level, NOS mRNA expression and ciliary beat frequency.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Child
  • Child, Preschool
  • Ciliary Motility Disorders / enzymology
  • Ciliary Motility Disorders / metabolism
  • Female
  • Gene Expression Regulation, Enzymologic*
  • Humans
  • Infant
  • Infant, Newborn
  • Kartagener Syndrome / enzymology*
  • Kartagener Syndrome / metabolism*
  • Leukocytes / cytology
  • Male
  • Nitric Oxide / metabolism*
  • Nitric Oxide Synthase / biosynthesis*
  • Nitric Oxide Synthase / metabolism
  • Nose / pathology
  • Polymorphism, Genetic
  • Protein Isoforms

Substances

  • Protein Isoforms
  • Nitric Oxide
  • Nitric Oxide Synthase