Focal transient ischemia increases APP-BP1 expression in neural progenitor cells

Neuroreport. 2011 Mar 30;22(5):200-5. doi: 10.1097/WNR.0b013e32834449cf.

Abstract

Amyloid precursor protein binding protein-1 (APP-BP1) binds to the carboxyl terminus of APP. In this study, we explored whether APP-BP1 expression is affected by focal transient cerebral ischemia induced by middle cerebral artery occlusion in Wistar rats. APP-BP1 expression was increased in the dentate gyrus of the hippocampus and in the subventricular zone of rats exposed to focal transient cerebral ischemia. In addition, APP-BP1 immunoreactivity overlapped with antidoublecortin and anti-5-bromo-2-deoxyuridine labeling. Focal transient cerebral ischemia has been reported earlier to induce neurogenesis in adult brains. The upregulation of APP-BP1 expression in neural progenitor cells after focal transient ischemia suggests that this protein contributes to the neurogenesis induced by transient ischemia and reperfusion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amyloid beta-Protein Precursor / metabolism
  • Animals
  • Carrier Proteins / biosynthesis*
  • DNA-Binding Proteins / biosynthesis*
  • Gene Expression
  • Gene Expression Profiling
  • Immunohistochemistry
  • Ischemic Attack, Transient / genetics
  • Ischemic Attack, Transient / metabolism*
  • Microscopy, Confocal
  • Neural Stem Cells / metabolism*
  • Neurogenesis / genetics*
  • Rats
  • Rats, Wistar
  • Ubiquitin-Activating Enzymes

Substances

  • Amyloid beta-Protein Precursor
  • Carrier Proteins
  • DNA-Binding Proteins
  • Nae1 protein, rat
  • Ubiquitin-Activating Enzymes