The modulation of myogenic cells differentiation using a semiconductor-muscle junction

Biomaterials. 2011 Jun;32(18):4228-37. doi: 10.1016/j.biomaterials.2011.02.024. Epub 2011 Mar 23.

Abstract

The present study is aimed to design a prototype of hybrid silicon-muscle cell junction, analog to an artificial neuromuscular junction prototype and relevant to the development of advanced neuro-prostheses and bionic systems. The device achieves focal Electric Capacitive Stimulation (ECS) by coupling of single cells and semiconductors, without electrochemical reaction with the substrate. A voltage change applied to a stimulation spot beneath an electrogenic cell leads to a capacitive current (charge accumulation) that opens voltage-gated ion channels in the membrane and generates an action potential. The myo-electronic junction was employed to chronically stimulate muscle cells via ECS and to induce cytosolic calcium transients in myotubes, fibers isolated from mouse FDB (fast [Ca(2+)](i) transients) and surprisingly also in undifferentiated myoblasts (slow [Ca(2+)](i) waves). The hybrid junction elicited, via chronic ECS, a differential reprogramming of single muscle cells by inducing early muscle contraction maturation and plasticity effects, such as NFAT-C3 nuclear translocation. In addition, in the presence of agrin, chronic ECS induced a modulation of AchR clustering which simulates in vitro synaptogenesis. This methodology can coordinate the myogenic differentiation, thus offering direct but non-invasive single cell/wiring, providing a platform for regenerative medicine strategies.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Action Potentials / physiology
  • Animals
  • Calcium / metabolism
  • Cell Differentiation / physiology*
  • Cells, Cultured
  • Electric Capacitance
  • Electric Stimulation / methods
  • Mice
  • Muscle Fibers, Skeletal / cytology
  • Muscle Fibers, Skeletal / metabolism
  • Muscle, Skeletal / cytology*
  • Muscle, Skeletal / physiology*
  • Myoblasts / cytology
  • Myoblasts / physiology
  • NFATC Transcription Factors / metabolism
  • Receptors, Cholinergic / metabolism
  • Semiconductors*
  • Silicon / metabolism

Substances

  • NFATC Transcription Factors
  • Receptors, Cholinergic
  • Calcium
  • Silicon