Expansion of CD16-negative natural killer cells in the peripheral blood of patients with metastatic melanoma

Clin Dev Immunol. 2011:2011:316314. doi: 10.1155/2011/316314. Epub 2011 Feb 23.

Abstract

Altered natural killer (NK) cell function is a component of the global immune dysregulation that occurs in advanced malignancies. Another condition associated with altered NK homeostasis is normal pregnancy, where robust infiltration with CD16- CD9+ NK cells can be identified in decidual tissues, along with a concomitant expansion of CD16- NK cells in the maternal peripheral blood. In metastatic melanoma, we identified a similar expansion of peripheral blood CD16- NK cells (median 7.4% in 41 patients with melanoma compared with 3.0% in 29 controls, P < .001). A subset of NK cells in melanoma patients also expresses CD9, which is characteristically expressed only on NK cells within the female reproductive tract. Expansion of CD16- NK cells was associated with elevated plasma transforming growth factor-beta (TGF-β levels (median 20 ng/ml, Spearman's ρ = 0.81, P = .015)). These findings suggest the possibility of exploring anti-TGF-β therapy to restore NK function in melanoma.

MeSH terms

  • Antigens, CD / analysis
  • Antigens, CD / immunology
  • Case-Control Studies
  • Female
  • Humans
  • Killer Cells, Natural / immunology
  • Killer Cells, Natural / metabolism*
  • Killer Cells, Natural / pathology
  • Male
  • Melanoma* / blood
  • Melanoma* / immunology
  • Melanoma* / pathology
  • Membrane Glycoproteins / analysis
  • Membrane Glycoproteins / immunology
  • Neoplasm Metastasis
  • Pregnancy
  • Receptors, IgG / deficiency
  • Receptors, IgG / immunology
  • Skin Neoplasms* / blood
  • Skin Neoplasms* / immunology
  • Skin Neoplasms* / pathology
  • Tetraspanin 29
  • Transforming Growth Factor beta / blood*
  • Transforming Growth Factor beta / immunology

Substances

  • Antigens, CD
  • CD9 protein, human
  • FCGR3A protein, human
  • Membrane Glycoproteins
  • Receptors, IgG
  • Tetraspanin 29
  • Transforming Growth Factor beta