[Gentiana veitchiorum particles inhibited LPS induced pulmonary alveolar macrophages (AM) TNF-α production and the underlying mechanism]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2011 Apr;27(4):364-6.
[Article in Chinese]

Abstract

Aim: To investigate the effect of Gentiana veitchiorum particles on the expression of TNF-α in pulmonary alveolar macrophages (AM) which induced by LPS, to explain the mechanism about anti-inflammatory action of Gentiana veitchiorum particles.

Methods: Purification of rat AM, TNF-α level in AM culture supernatant was detected by ELISA. Western blot method for detecting the expression of TNF-α and pERK in the AM. While application of ERK antagonist (PD98059) in rat AM and the expression of TNF-α was observed by Western blot.

Results: Gentiana veitchiorum particles can reduce the LPS induced AM TNF-α increase in dose dependent manner. Gentiana veitchiorum particles (100 mg/L) can significantly reduce the LPS induced pERK and TNF-α protein expression in AM. compared with LPS stimulation group, we found that ERK inhibitor (PD98059 30 mol/L), Gentiana veitchiorum particles intervention and Gentiana veitchiorum particles+PD98059 groups' TNF-α expression were significantly reduced in rat AM.

Conclusion: Gentiana veitchiorum particles can inhibit the LPS induced pulmonary AM TNF-α expression, one of the possible mechanism is to inhibit the extracellular signal transduction pathway.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Extracellular Signal-Regulated MAP Kinases / metabolism
  • Gentiana*
  • Lipopolysaccharides / antagonists & inhibitors*
  • MAP Kinase Signaling System / physiology
  • Macrophages, Alveolar / drug effects*
  • Macrophages, Alveolar / immunology
  • Male
  • Phosphorylation
  • Rats
  • Rats, Sprague-Dawley
  • Tumor Necrosis Factor-alpha / biosynthesis*

Substances

  • Lipopolysaccharides
  • Tumor Necrosis Factor-alpha
  • Extracellular Signal-Regulated MAP Kinases