Role of interleukin 28B rs12979860 C/T polymorphism on the histological outcome of chronic hepatitis C: relationship with gender and viral genotype

J Clin Immunol. 2011 Oct;31(5):891-9. doi: 10.1007/s10875-011-9547-1. Epub 2011 Jun 7.

Abstract

Background: This study aimed to determine whether the single-nucleotide polymorphism (rs12979860 C/T) of the interleukin 28B (IL-28B) gene, which is associated with hepatitis C virus (HCV) clearance, is also associated with fibrosis in chronic HCV infection.

Methods: An RFLP-PCR technique was used to genotype 629 HCV-positive patients (200 with cirrhosis) and 428 healthy control subjects.

Results: The genotype frequencies in the controls and chronic hepatitis C patients were as follows: C/C 47.0% vs. 32.6%, C/T 41.8% vs. 52.8% and T/T 11.2% vs. 14.6% (p < 0.0001). The C allele frequency was higher in HCV-2- (0.635) and 3- (0.692) infected patients in comparison to those infected with HCV-1 (0.550) or 4-5 (0.600) (p < 0.001). Infected T/T homozygotes had a mean staging score higher than other patients (3.50 vs. 3.04, p < 0.05).

Conclusions: IL-28B rs12979860 C/T polymorphism is associated with a greater likelihood of HCV persistence, particularly in HCV genotypes 1 and 4. The T allele affects the severity of liver fibrosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • DNA Mutational Analysis
  • DNA, Viral / analysis*
  • Disease Progression
  • Female
  • Fibrosis
  • Gene Frequency
  • Genetic Association Studies
  • Genotype
  • Hepacivirus / pathogenicity
  • Hepacivirus / physiology*
  • Hepatitis C, Chronic / epidemiology
  • Hepatitis C, Chronic / genetics*
  • Hepatitis C, Chronic / immunology
  • Hepatitis C, Chronic / physiopathology
  • Humans
  • Interferons
  • Interleukins / genetics*
  • Liver / pathology
  • Male
  • Middle Aged
  • Polymorphism, Single Nucleotide
  • Retrospective Studies
  • Risk
  • Sex Factors*
  • Viral Load / genetics

Substances

  • DNA, Viral
  • interferon-lambda, human
  • Interleukins
  • Interferons