Pancreatic exocrine and gallbladder function during long-term treatment with octreotide (SMS 201-995)

Digestion. 1990:45 Suppl 1:72-6. doi: 10.1159/000200266.

Abstract

Since octreotide (SMS 201-995, Sandostatin; Sandoz Pharmaceuticals) is a potent inhibitor of pancreatic exocrine secretion and gallbladder contraction, long-term treatment with this drug may theoretically result in impaired pancreatic function and gallstones. However, we observed excellent pancreatic exocrine function--as assessed by the PABA/PAS test--in acromegalics who received octreotide treatment for more than 6 months. Plasma cholecystokinin showed a significant, although blunted, postprandial response, which exceeded the threshold for gallbladder contraction in healthy controls. Remarkably, postprandial gallbladder contraction was completely abolished for at least 2 h during octreotide treatment. In contrast to other studies, none of 16 acromegalic patients on long-term octreotide treatment developed gallstones. Although the incidence of gallstones in patients on long-term octreotide treatment may be increased, the risk seems to be variable.

Publication types

  • Review

MeSH terms

  • 4-Aminobenzoic Acid / urine
  • Acromegaly / drug therapy*
  • Aminosalicylic Acid / urine
  • Cholecystokinin / blood
  • Gallbladder / drug effects*
  • Humans
  • Octreotide / therapeutic use*
  • Pancreas / drug effects*

Substances

  • Aminosalicylic Acid
  • Cholecystokinin
  • Octreotide
  • 4-Aminobenzoic Acid