Endothelial dysfunction as assessed by flow-mediated dilation in patients with cardiac syndrome X: role of inflammation

Eur Rev Med Pharmacol Sci. 2011 Sep;15(9):1074-7.

Abstract

Background: Endothelial dysfunction, reduced coronary flow reserve and increased markers of inflammation are detectable in cardiac syndrome X (CSX). In this study we investigated the relation between inflammation and systemic endothelial function in CSX patients.

Methods: We studied 42 CSX patients (55 +/- 6 years, 14 men) and 20 healthy subjects (52 +/- 7 years, 9 men). Systemic endothelial function was assessed by flow-mediated dilation (FMD) of the brachial artery after 5-minute of forearm cuff inflation. Serum C-reactive protein (CRP) was measured by a high-sensitivity method.

Results: FMD was significantly lower in CSX patients compared to controls (4.8 +/- 4.4 vs. 13.7 +/- 4%, p < 0.001), whereas CRP levels were higher in CSX patients than in controls (2.7 +/- 2.4 vs. 0.7 +/- 0.4 mg/L, p = 0.001). In CSX patients FMD showed a significant inverse correlation with CRP levels, even after adjustment for potentially confounding variables (r = -0.34, p = 0.006).

Conclusion: An impaired FMD is detectable in CSX patients, suggesting a generalized abnormality in vascular function. Subclinical inflammation se is to play a significant role in the impairment of endothelium-dependent vasodilator function of these patients.

MeSH terms

  • Brachial Artery / diagnostic imaging
  • Brachial Artery / physiopathology*
  • C-Reactive Protein / analysis*
  • Case-Control Studies
  • Endothelium, Vascular / diagnostic imaging
  • Endothelium, Vascular / physiopathology*
  • Female
  • Humans
  • Inflammation / diagnostic imaging
  • Inflammation / immunology
  • Inflammation / physiopathology*
  • Inflammation Mediators / blood*
  • Italy
  • Male
  • Microvascular Angina / diagnostic imaging
  • Microvascular Angina / immunology
  • Microvascular Angina / physiopathology*
  • Middle Aged
  • Ultrasonography, Doppler, Pulsed
  • Up-Regulation
  • Vasodilation*

Substances

  • Inflammation Mediators
  • C-Reactive Protein