Histopathological changes induced by Hemiscorpius lepturus scorpion venom in mice

Toxicon. 2012 Mar 1;59(3):373-8. doi: 10.1016/j.toxicon.2011.12.011. Epub 2011 Dec 31.

Abstract

Envenomation by Hemiscorpius lepturus (H. lepturus) is associated with local necrosis, followed by systemic manifestations. In this work the LD₅₀ of H. lepturus venom were determined by subcutaneous (SC) injection in white Balb/c mice (5 mg/kg). Histopathological alterations in organs such as kidney, heart, liver, lungs, stomach and intestine were determined in 3, 6, 12 and 24 h following experimental (SC) envenoming injection of one LD ₅₀ of the venom in Balb/c mice. Histological studies showed degenerative changes in the kidney with disorganized glomeruli and necrotic tubular in 3 h and reached to its climax in 6 h. Myocardium showed massive myocytolysis with interstitial necrosis in 3 h and reached to its peak after 6 h past envenoming. Bowels showed edema of lamina propria and slight villous necrosis. The enzymatic activities of creatine kinase (CK) and lactate dehydrogenase (LDH) were significantly increased in the serum in 9 h. No necrotic lesion observed in lungs and liver. The results indicate that the venom of H. lepturus is a highly cytotoxic, and induces massive tissue damages in specific organs, starting from the heart and kidney as the first target in 3 h and ends to the bowels in 6 h post envenomation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Creatine Kinase / blood
  • Gastrointestinal Tract / drug effects
  • Gastrointestinal Tract / pathology*
  • Heart / drug effects
  • Heart / physiopathology
  • Injections, Subcutaneous
  • Kidney / drug effects
  • Kidney / pathology*
  • L-Lactate Dehydrogenase / blood
  • Lethal Dose 50
  • Liver / drug effects
  • Liver / pathology
  • Lung / drug effects
  • Lung / pathology
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Myocardium / pathology*
  • Scorpion Venoms / toxicity*
  • Scorpions

Substances

  • Scorpion Venoms
  • L-Lactate Dehydrogenase
  • Creatine Kinase