Downregulation of Id1 by small interfering RNA in gastric cancer inhibits cell growth via the Akt pathway

Mol Med Rep. 2012 Apr;5(4):1075-9. doi: 10.3892/mmr.2012.749. Epub 2012 Jan 12.

Abstract

Inhibitor of differentiation or DNA binding (Id1) is a member of the helix-loop-helix transcription factor family that is overexpressed in various types of cancer, including gastric carcinoma. Previous studies showed that Id1 is a prognostic marker in patients with gastric cancer. However, the role of Id1 in the proliferation of human gastric cancer cells has yet to be clarified. In the present study, we downregulated the Id1 gene in SGC-7901 gastric cancer cells by RNA interference, and we also constructed a recombinant plasmid-expressing Id1 to investigate its effects on the proliferation of SGC-7901 cells. Results showed that the downregulation of Id1 inhibited proliferation of SGC-7901 cells, while the upregulation of Id1 had no effect on SGC-7901 cell proliferation. The potential mechanism was also investigated. The changes of certain proteins associated with cell proliferation, apoptosis and the cell cycle were detected by western blotting. Furthermore, we demonstrated a positive correlation between Id1 and phospho-Akt expression in SGC-7901 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Apoptosis
  • Cell Division
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Down-Regulation*
  • G2 Phase
  • Humans
  • Inhibitor of Differentiation Protein 1 / antagonists & inhibitors*
  • Inhibitor of Differentiation Protein 1 / genetics
  • Inhibitor of Differentiation Protein 1 / metabolism
  • Proto-Oncogene Proteins c-akt / metabolism*
  • RNA Interference*
  • RNA, Small Interfering / pharmacology*
  • Signal Transduction
  • Stomach Neoplasms / metabolism

Substances

  • Antineoplastic Agents
  • ID1 protein, human
  • Inhibitor of Differentiation Protein 1
  • RNA, Small Interfering
  • Proto-Oncogene Proteins c-akt