Abstract
Recent data suggest that chronic increment of reactive oxygen species (ROS) may be involved in the development and progression of chronic prostatic disease, such as BPH and PCa; adipose tissue produces bioactive substances called adipokines, also involved in the production of ROS. Our study aims to evaluate the relationship between obesity and oxidative stress in prostate disease.
MeSH terms
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Adenocarcinoma / chemistry
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Adenocarcinoma / complications*
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Adipokines / metabolism
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Adipose Tissue / metabolism
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Adiposity
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Aged
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Biopsy
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Body Mass Index
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Disease Progression
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Glutathione / analysis
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Humans
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Male
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Middle Aged
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Obesity / complications*
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Obesity / metabolism
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Organ Size
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Oxidative Stress*
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Plethysmography, Whole Body
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Prostate / chemistry
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Prostate / pathology
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Prostate-Specific Antigen / analysis
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Prostatic Hyperplasia / complications*
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Prostatic Hyperplasia / metabolism
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Prostatic Neoplasms / chemistry
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Prostatic Neoplasms / complications*
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Prostatitis / complications*
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Prostatitis / metabolism
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Reactive Oxygen Species / metabolism
Substances
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Adipokines
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Reactive Oxygen Species
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Prostate-Specific Antigen
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Glutathione